Should humans with Crohn’s disease be reclassified as having Johne’s disease? Enjoy the latest commentary from Dr. William C. Davis of Washington State University, whose lab will be presenting their latest research at the Berkeley Conference on September 21st!
Nontuberculous mycobacteria (NTM) are not only conventional opportunistic pathogens, but they are important sources of triggers of chronic immune-mediated and autoimmune disorders. In this short note and associated paper, Dr. Karel Hruska highlights the risk posed by NTM in water, food, and soil. Hence NTM are associated with the so called “civilization diseases” (Type 1 diabetes, Crohn’s disease, multiple sclerosis, asthma, psoriasis, rheumatoid arthritis, and others.)
The 2018 Human Para conference, Mycobacterial Implications in Crohn’s and Chronic Inflammatory Diseases, is only weeks away! As an introduction to MAP, the conference, and our foundation, Dr. Michael Collins has provided a short video summarizing the issues and setting the stage.
The author of this recent case study, Branko G. Celler, an Emeritus Professor at the University of New South Wales in Sydney, Australia, developed a particularly serious form of sarcoidosis in 2012. While sarcoidosis is a chronic disease characterized by the formation of granulomas in various organs, only a small portion of patients have cardiac involvement, which can lead to symptomatic conduction disturbances, ventricular arrhythmias, progressive heart failure and sudden death. Read more about his recovery with anti-mycobacterial antibiotics.
Have you reserved your spot for the Human Para Conference, Mycobacterial Implications in Crohn’s and Chronic Inflammatory Diseases, coming to Berkeley, CA on September 21st & 22nd? It’s not too late! Registration closes a week prior to the event, and a detailed agenda, abstracts and updated registration information have recently been added to the conference pages. Additionally, we are pleased to announce that portions of this activity have been approved for AMA PRA Category 1 CreditTM.